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T-cell CX3CR1 expression as a dynamic blood-based biomarker of response to immune checkpoint inhibitors

  • Takayoshi Yamauchi
  • , Toshifumi Hoki
  • , Takaaki Oba
  • , Vaibhav Jain
  • , Hongbin Chen
  • , Kristopher Attwood
  • , Sebastiano Battaglia
  • , Saby George
  • , Gurkamal Chatta
  • , Igor Puzanov
  • , Carl Morrison
  • , Kunle Odunsi
  • , Brahm H. Segal
  • , Grace K. Dy
  • , Marc S. Ernstoff
  • , Fumito Ito
  • Roswell Park Cancer Institute
  • Merck
  • SUNY Buffalo
  • The University of Chicago
  • National Institutes of Health

Research output: Contribution to journalArticlepeer-review

149 Scopus citations

Abstract

Immune checkpoint inhibitors (ICI) have revolutionized treatment for various cancers; however, durable response is limited to only a subset of patients. Discovery of blood-based biomarkers that reflect dynamic change of the tumor microenvironment, and predict response to ICI, will markedly improve current treatment regimens. Here, we investigate CX3C chemokine receptor 1 (CX3CR1), a marker of T-cell differentiation, as a predictive correlate of response to ICI therapy. Successful treatment of tumor-bearing mice with ICI increases the frequency and T-cell receptor clonality of the peripheral CX3CR1+CD8+ T-cell subset that includes an enriched repertoire of tumor-specific and tumor-infiltrating CD8+ T cells. Furthermore, an increase in the frequency of the CX3CR1+ subset in circulating CD8+ T cells early after initiation of anti-PD-1 therapy correlates with response and survival in patients with non-small cell lung cancer. Collectively, these data support T-cell CX3CR1 expression as a blood-based dynamic early on-treatment predictor of response to ICI therapy.

Original languageEnglish
Article number1402
JournalNature Communications
Volume12
Issue number1
DOIs
StatePublished - Dec 1 2021

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