Proliferative retinopathies in children

Philip J. Ferrone; Steven Awner

doi:10.1007/978-3-642-12041-1_15

Proliferative retinopathies in children

Philip J. Ferrone
, Steven Awner

Department of Ophthalmology

Columbia University

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

3 Scopus citations

Abstract

The first two entities, diabetes and SC, develop retinopathy based upon a systemic metabolic disruption of retinal physiology. The other proliferative retinopathies, except for chronic retinal detachment, are due to either genetic or developmental abnormalities. They all exert their deleterious effects by inducing ischemia and thereby disrupting the metabolic needs of the retina. This invariably leads to neovascularization (NV) of the retina with resultant glial organization with or without hemorrhage into the vitreous. Subsequent contraction of the vitreous gel causes retinal detachment and often blindness.

Original language	English
Title of host publication	Pediatric Retina
Publisher	Springer Berlin Heidelberg
Pages	351-360
Number of pages	10
ISBN (Print)	9783642120404
DOIs	https://doi.org/10.1007/978-3-642-12041-1_15
State	Published - 2011

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10.1007/978-3-642-12041-1_15

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title = "Proliferative retinopathies in children",

abstract = "The first two entities, diabetes and SC, develop retinopathy based upon a systemic metabolic disruption of retinal physiology. The other proliferative retinopathies, except for chronic retinal detachment, are due to either genetic or developmental abnormalities. They all exert their deleterious effects by inducing ischemia and thereby disrupting the metabolic needs of the retina. This invariably leads to neovascularization (NV) of the retina with resultant glial organization with or without hemorrhage into the vitreous. Subsequent contraction of the vitreous gel causes retinal detachment and often blindness.",

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N2 - The first two entities, diabetes and SC, develop retinopathy based upon a systemic metabolic disruption of retinal physiology. The other proliferative retinopathies, except for chronic retinal detachment, are due to either genetic or developmental abnormalities. They all exert their deleterious effects by inducing ischemia and thereby disrupting the metabolic needs of the retina. This invariably leads to neovascularization (NV) of the retina with resultant glial organization with or without hemorrhage into the vitreous. Subsequent contraction of the vitreous gel causes retinal detachment and often blindness.

AB - The first two entities, diabetes and SC, develop retinopathy based upon a systemic metabolic disruption of retinal physiology. The other proliferative retinopathies, except for chronic retinal detachment, are due to either genetic or developmental abnormalities. They all exert their deleterious effects by inducing ischemia and thereby disrupting the metabolic needs of the retina. This invariably leads to neovascularization (NV) of the retina with resultant glial organization with or without hemorrhage into the vitreous. Subsequent contraction of the vitreous gel causes retinal detachment and often blindness.

UR - https://www.scopus.com/pages/publications/84888070681

U2 - 10.1007/978-3-642-12041-1_15

DO - 10.1007/978-3-642-12041-1_15

M3 - Chapter

AN - SCOPUS:84888070681

SN - 9783642120404

SP - 351

EP - 360

BT - Pediatric Retina

PB - Springer Berlin Heidelberg

ER -

Proliferative retinopathies in children

Abstract

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